Most importantly, we’ve connected these observations to hnRNP K function simply by showing the fact that 4A mutant hnRNP K proteins no more promotes p53- and DNA damage-dependent induction of transcription through the gene promoter and in addition will not foster DNA damage-induced binding of p53 towards the and most likely also certain various other p53 focus on genes

Most importantly, we’ve connected these observations to hnRNP K function simply by showing the fact that 4A mutant hnRNP K…

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